• Pseudomonas aeruginosa in co-culture

    Structural component of bacterial targeting type VI secretion system P. aeruginosa (green) assembled in presence of competitor organism (red).

    Courtesy of: Mougous Lab

  • Bacteriophage in biofilm

    Filamentous bacteriophage organizing the biofilm matrix into a liquid crystal
    Courtesy: Singh Lab

     

     

  • Inflammatory Death of Mouse Macrophages

    Mouse macrophages undergoing pyroptosis.

    Courtesy of: Cookson Lab

  • Pseudomonas aeruginosa

    Colonies of Pseudomonas aeruginosa expressing different amounts of exopolysaccharides.

    Courtesy of: Harwood Lab

  • HIV-1 Envelope Evolution

    Reconstructed phylogenetic network of HIV-1 envelope sequences (C2-V5) from subtype B.

    Courtesy of: Mullins Lab

  • YopM Crystal Structure

    Crystal structure of the Yersinia virulence protein YopM

    Courtesy of: Cookson Lab

  • Emergence of Highly Pathogenic Clones of Escherichia coli

    Genetic typing of uropathogeic E. coli reveals strong association of some clones with high sensitivity or extreme resistance to multiple antibiotics.

    Courtesy: Sokurenko Lab

  • Quorum sensing in Vibrio fischeri

    Lux genes coding for light production are activated by quorum sensing at high cell density. The light produced by the bacteria exposed the film for the image.
     
    Courtesy: Greenberg Lab

     

     

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Latest News

Congratulations to Dr. Joseph Mougous on his HHMI Investigator status renewed for another 7 years! Professor Mougous, who became an HHMI investigator in 2015 runs a research lab making ground-breaking discoveries in microbiology.  The lab focuses on secretion systems that deliver toxic proteins to neighboring bacterial cells, bacterial interactions in the human gut, novel toxin activities in diverse bacteria, and secreted virulence factors that subvert host cell defenses and facilitate infection. More on Dr. Mougous’ work can be found here.

Scientists in Washington researching COVID-19 Omicron variant to help protect public

"The alarming thing is we didn’t see a lot of these mutations in related viruses. Usually viruses accumulate mutations over time, and so we could kind of track as mutations emerge, and we see more and more over time. But there was this gap, and as soon Omicron was sequenced, all of these new mutations popped up. So, there’s a lot of questions going around as to how that happened," said Dr. Jesse Erasmus, acting assistant professor at UW Medicine.

The Department of Microbiology is also conducting a second search for a full-time (12 month) Assistant Teaching Professor.  If interested, apply here.

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